Lab Report Summary Questions Module 5.do...
Lab_Report_Summary_Questions_Module_5.docx.docx-Lab Report Summary Quesons: 1. In
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Lab_Report_Summary_Questions_Module_5.docx.docx-Lab Report Summary Quesons: 1. In
Lab Report Summary Questions Module...
Lab_Report_Summary_Questions_Module_5.docx.docx-Lab Report Summary Quesons: 1. In
Page 1
Lab Report Summary Quesons:
1.
In exercise 1, scenario 1 (cysc fibrosis), what is the genotype of the mother and father?
Mother: Ff
Father: Ff
2.
In exercise 1, scenario 1 (cysc fibrosis), would the odds of having a second cysc
fibrosis child change if the couple’s first child were normal? Why or why not?
75% because it increases every me they have a child that has not had the gene.
3.
In exercise 2, how would the pedigree for hemophilia change if it were carried on the Y
chromosome?
There would be a 2:3 rao to passing it onto the children
4.
Considering exercise 3, what would the karyotype of a child with trisomy 22 look like?
What syndrome would this child have?
hps://synapse.koreamed.org/ArcleImage/0201EMJ/emj-35-62-g001-l.jpg
Deleon Syndrome
5.
Imagine that a human egg with no sex chromosomes is ferlized by a normal Y carrying
sperm, what would the karyotype look like? Research this syndrome and see if there are
any living examples of this type of ferlizaon event represented in live births. What do
you think explains what you have found?


Page 2
From what I have read and looked at, I could not find any documented cases of the child
living. This explains that because this is sex oriented is meaning this child does not
develop the proper organs to sustain life.


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